DISPLASIA DENTINARIA PDF

March 30, 2019 posted by

7 nov. CONCEITO: TIPOS DE DISPLASIA DENTÁRIA TIPO I UNIVERSIDADE PAULISTA-UNIP INSTITUTO DE CIÊNCIAS DA SAÚDE-ICS. A displasia dentinária é uma alteração do desenvolvimento da dentina rara, de origem autossômica dominante, o qual é geralmente dividida em 2 tipos. A displasia fibrosa é lesão pseudo-neoplásica de etiologia desconhecida, da pirâmide nasal à esquerda, deslocamento da arcada dentária superior para a.

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Dentin dysplasia

The esthetic and functional goals proposed at the onset of treatment were achieved, due to the meticulous use of orthodontic mechanics, radiographic monitoring at various stages of the treatment and longer intervals between clinical sessions. Right to privacy and informed consent. In this case, the patient presented permanent dentition with normal root development on 3 hemi-arches. Retrograde fillings and periapical surgery is a treatment option for teeth with longer roots, as well as orthodontic treatment.

A possible biological basis can be understood from detninaria regulatory role of the Nfic gene. In other projects Wikimedia Commons. Oral and maxillofacial pathology. Scand J Dent Res.

Eur J Oral Sci. Nature and frequency of dental changes in idiopathic hypoparathyroidism and pseudohypoparathyroidism. The term short root anomaly SRA12,19 also known as root dwarfism, 27 was introduced and first described by Lind, 19 into categorize a rare developmental anomaly characterized by full root formation, but with genetically determined foreshortening. The final extraoral radiographs and cephalometric superimposition are shown in Figures 21 and Panoramic radiographic examination confirmed that teeth 36 and 46 were missing and showed that teeth 34, 35 and 37 presented alteration in the pattern of root formation compared to the contralateral teeth Fig.

Dentinogenic ghost cell tumour DGCT. Midtbo M, Halse A. Most DD1 cases have autosomal dominant inheritance, which means that individuals of the same family may be affected 2, Alves 1Fernando Canavarros 2Daniela S. Due to Fibrous Dysplasia and Ossifying Fibroma similar clinical courses, the histopathological findings are essential to their differential diagnosis. The intraoral evaluation showed good dental health and healthy periodontal tissues.

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The main differential diagnosis of the monostotic form on head and neck bones is Ossifying Fibroma which some consider another form of the same entity. Frequent findings include delayed dental eruption pattern 13,14opaque incisal borders 14spontaneous premature exfoliation 14because of the short, blunt, tapering, or even absent roots 1and discomfort caused by the severe tooth mobility, especially after meals 2,8,9.

Ghost cells were seen trapped in the dysplastic dentin and mitosis could not be observed Fig. An alternative to conventional therapy would be retrograde filling and periapical curettage. Behavior of short-rooted teeth in the upper bilateral central incisors. Extraction space was then closed and orthodontic detailing performed with the inclusion of second molars and individual bends and torques in the 0.

Orthodontic movement of teeth with short root anomaly: should it be avoided, faced or ignored?

Intraosseous dentinogenic ghost cell tumor: Irrespective of treatment options, patient monitoring should be inexcusably undertaken in order to detect displassia or re-sprouting. Unsourced material may be challenged and removed. The examination of the extraoral radiographs confirmed the absence of significant skeletal asymmetry and the agenesis of the second lower left and right premolars 35 and 45as well as the lower right third molar 48 Fig All of them had recurrences after surgical removal, diagnosed between first and eighth year of follow up.

Radiographic examination is an important diagnostic tool for identifying this dental anomaly 12revealing short, blunt and malformed roots, similar to those observed in taurodontic teeth, obliteration of all pulp chambers with crescent-shaped pulp remnants parallel to the cementoenamel junction and several periapical radiolucencies in noncarious teeth 1,2,5,7,8, Journal of Dental Research.

Correction of midline deviations was aided by open coil springs, anterior retraction and inferior reciprocal mechanics to close the agenesis space of the left second lower premolar 35 Figs 13 and On the other hand, according to Wesley et al. August 11, Revised and accepted: The second deciduous left lower molar 75 was still in place Fig.

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Travesera de Gracia,Barcelona, Barcelona, ES, maxilo elsevier. The calcifying odontogenic cyst: Thus, SRA differs from other conditions characterized by short roots. A review of ninety-two cases with re-evaluation of their dentinaira as cysts or neoplasms, the nature of ghost cells and subclassification. Clinical examination disclosed grade III mobility of teeth [] with rhizolysis of teeth 41, 31 and 32, and painless swelling from the right lateral incisive of the jaw to the left second molar area of the jaw.

Ethical approval Not required. Dentin dysplasia type I: J Oral Maxillofac Pathol. Ann Otol Rhinol Laryngol ; This page was last edited on 24 Novemberat By using this site, you agree to the Terms of Use and Privacy Policy. Dentin dysplasia type I”.

Int J Paediatr Dent. Dentinogenic ghost cell tumors. Radiologic and genetic perspectives in a six-generation family”. However this new classification supposed a self-contradiction since the lesion was again classified as “benign odontogenic tumor”.

Leed e Seaman, emidentificaram o osso frontal e o esfenoidal como os mais comumente envolvidos 4. December 03, The authors report no commercial, proprietary or financial interest in the products or companies described in this article.

An unusual combination of idiopathic generalized short-root anomaly associated with microdontia, taurodontia, multiple dens invaginatus, obliterated pulp chambers and infected cyst: The clinical and radiographic findings of this case led us to suspect of a variation of DD1, since some of the typical signs of this dental developmental abnormality, such as pulp chamber obliteration and periapical radiolucencies associated with sound teeth 2,5,14,16were not present.

The presentation of case reports in which classic signs of DD1 are found, but other typical features are not present highlights the clinical and radiographic variations of DD1.